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This phenomenon can be attributed to the body’s limited capacity to utilise L-arginine for NO synthesis in specific conditions
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Accordingly, it is hypothesized that lower levels of prostaglandins in seminal plasma, including prostasomes, might disrupt the typical antiplatelet and anti‐inflammatory effects, leading to an increased risk of thrombotic events in the context of pre‐eclampsia (García‐Montalvo et al
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