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The main sources of reactive oxygen species (ROS) in diabetic... | Download Scientific Diagram
Mechanistically, S100P orchestrates acetyl-CoA carboxylase 1 (ACC1)-dependent lipid metabolic reprogramming to promote the accumulation of polyunsaturated fatty acid (PUFA)-derived phospholipids in HCC cells, thereby enhancing the generation of lipid peroxidation byproducts
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In vivo rat metabolism and pharmacokinetic studies of ginsenoside Rg3
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