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Wang, Fibroblasts and myofibroblasts in wound healing: force generation and measurement

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Moreover, chronic hyperglycemia leads to increased PKC activation via increased de novo synthesis of DAG, which activates multiple PKC isoforms in diabetic platelets, particularly PKCβ2 and PKCδ (Geraldes and King, 2010), where upregulation of PKCβ2 has been linked to microvascular complications such as retinopathy and nephropathy, and consequent PKCβ inhibition and insulin treatment have been shown to reduce platelet hyperactivity (Kimura et al., 2001)

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