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Our results showed that LPS stimulation markedly enhanced the phosphorylation of p38, JNK and ERK in Caco-2 cells, while 5 μg/mL GA weakened the LPS-induced phosphorylation of p38, JNK and ERK ( Figure 7), indicating that MAPK is involved in the regulatory effect of GA on the inhibition of intestinal inflammation induced by LPS

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